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Am I right in thinking that one could dramatically increase serum levels of N-arachidonoylethanolamine (anandamide) - anandamide especially but other endocannabinoids as well - by consuming the following readily obtainable substances: Ibuprofen (inhibits cyclooxygenase which converts arachidonic acid into various prostanoids.) Linoleic acid (converted into arachidonic acid) Lecithin (consists of: phosphatidylethanolamine, phosphatidylcholine, phosphatidylinositol, phosphatidic acid) Inositol 5-lipoxygenase inhibitor (Zileuton/Azelastine/Diethylcarbamazine/nordihydroguaiaretic acid) (arachidonate 5-lipoxygenase converts arachidonic acid into leukotrienes) Or do you suppose that fatty acid amide hydrolase is too abundant or too efficient for this combination to have a noticeable psychotropic effect?

If so, are you aware of any fatty acid amide hydrolase inhibitors that can be obtained with relative facility? Cocoa may contain alkaloids that inhibit the enzyme that breaks down anandamine.

Stress can increase endocannabinoids, and so can lowered levels of leptin and elevated calcium.

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